Fuel metabolism in pregnancy

فهرست عناوین اصلی در این پاورپوینت

فهرست عناوین اصلی در این پاورپوینت

● Plan of presentation
● Introduction
● Fuel metabolism in pregnancy
● Glucose metabolism in pregnancy
● Magnitude of problem: Global
● Magnitude of the problem – India
● Risk factors
● Maternal complications
● Maternofetal complications
● Neonatal complications
● Congenital anomalies
● Whom to screen ?
● High risk
● When and how to screen?
● 50 g GTT
● 100 g GTT
● 75 g GTT
● Whom and when to screen? Indian Scenario -The DIPSI Guidelines
● MANAGEMENT ISSUES
● Medical nutrition therapy
● Calorie allotment
● Carb intake
● Calorie distribution
● Monitoring BG
● Glycemic targets (ACOG)
● PHARMACOLOGICAL INTERVENTION
● Insulin
● OHA in pregnancy
● Fetal monitoring
● Timing of delivery
● Management of labor and delivery
● Glycemic management during labour
● Post partum follow up
● Immediate management of neonate
● Management of neonate
● Future risks – Mother
● Who will progress to DM?
● Preconception counselling
● Risk of developing DM in offspring
● Conclusion

نوع زبان: انگلیسی حجم: 1.35 مگا بایت
نوع فایل: اسلاید پاورپوینت تعداد اسلایدها: 65 صفحه
سطح مطلب: نامشخص پسوند فایل: ppt
گروه موضوعی: زمان استخراج مطلب: 2019/05/16 05:16:58

لینک دانلود رایگان لینک دانلود کمکی

اسلایدهای پاورپوینت مرتبط در پایین صفحه

عبارات مهم استفاده شده در این مطلب

عبارات مهم استفاده شده در این مطلب

–, pregnancy, ., insulin, glucose, risk, percent, syndrome, gdm, screening, metabolism, type,

توجه: این مطلب در تاریخ 2019/05/16 05:16:58 به صورت خودکار از فضای وب آشکار توسط موتور جستجوی پاورپوینت جمع آوری شده است و در صورت اعلام عدم رضایت تهیه کننده ی آن، طبق قوانین سایت از روی وب گاه حذف خواهد شد. این مطلب از وب سایت زیر استخراج شده است و مسئولیت انتشار آن با منبع اصلی است.

https://www.cmcendovellore.org/downloads/diabetes-pregnancy.ppt

در صورتی که محتوای فایل ارائه شده با عنوان مطلب سازگار نبود یا مطلب مذکور خلاف قوانین کشور بود لطفا در بخش دیدگاه (در پایین صفحه) به ما اطلاع دهید تا بعد از بررسی در کوتاه ترین زمان نسبت به حدف با اصلاح آن اقدام نماییم. جهت جستجوی پاورپوینت های بیشتر بر روی اینجا کلیک کنید.

عبارات پرتکرار و مهم در این اسلاید عبارتند از: –, pregnancy, ., insulin, glucose, risk, percent, syndrome, gdm, screening, metabolism, type,

مشاهده محتوای متنیِ این اسلاید ppt

مشاهده محتوای متنیِ این اسلاید ppt

dr. kanakamani madhivanan m.d. d.m. endocrinology assistant professor department of endocrinology diabetes metabolism christian medical college vellore plan of presentation introduction physiology of fuel metabolism in normal pregnancy pathophysiology of gdm epidemiology of gdm screening and diagnosis maternal and fetal risks management of gdm obstetric management introduction global increase in prevalence of dm individual importance hyperglycemia in pregnancy has adverse effects on both mother and fetus public health importance – rising epidemic of dm in part attributed to the diabetic pregnancies prevention of type ۲ dm should start intrauterine and continue throughout life introduction gestational diabetes gdm is defined as any degree of impaired glucose tolerance of with onset or first recognition during pregnancy . many are denovo pregnancy induced some are type ۲ ۳۵ ۴ ۱ have antibodies introduction difficult to distinguish pregestational type ۲ dm and denovo gdm fasting hyperglycemia blood glucose greater than ۱۸ mg dl on ogt acanthosis nicgrans hba۱c ۵.۳ a systolic bp ۱۱ mm hg bmi ۳ kg m۲ fetal anomalies clues for type ۱ lean dka during pregnancy severe hyperglycemia with large doses of insulin fuel metabolism in pregnancy goal is uninterrupted nutrient supply to fetus the metabolic goals of pregnancy are ۱ in early pregnancy to develop anabolic stores to meet metabolic demands in late pregnancy ۲ in late pregnancy to provide fuels for fetal growth and energy needs. glucose metabolism in pregnancy early pregnancy e۲ prl stimulates b cells –insulin sensitivity same and peripheral glucose utilisation – ۱ fall in bg levels late pregnancy fetoplacental unit extracts glucose and aminoacids fat is used mainly for fuel metabolism insulin sensitivity decreases progressively upto ۵ ۸ during the third trimester variety of hormones secreted by the placenta especially hpl and placental growth hormone variant cortisol prl e۲ and prog glucose metabolism in pregnancy fetus fat glucose aminoacids insulin resistance hyperinsulinemia fasting accelerated starvation and esxaggerated ketosis maternal hypoglycemia hypoinsulinemia hyperlipidemia and hyperketonemia fed hyperglycemia hyperinsulinemia hyperlipidemia and reduced tissue sensitivity to insulin ۲۴ hour insulin requirement before conception is approximately .۸ units kg. in the first trimester the insulin requirement rises to .۷units kg of the pregnant weight – more unstable glycemia with a tendency to low fasting plasma glucose and high postprandial excursions and the occurrence of nocturnal hypoglycemia by the second trimester the insulin requirement is .۸ units per kilogram. from ۲۴th month onwards steady increase in insulin requirement and glycemia stabilises by third trimester the insulin requirement is .۹ ۱. unit kg pregnant weight per day last month – may be a decrease in insulin and hypoglycemias esp. nocturnal magnitude of problem global prevalence of gdm varies worldwide and among different racial and ethnic groups within a country america – white women ۳.۹ and asian ۸.۷ europe – .۶ to ۳.۶ australia – ۳.۶ to ۴.۷ indian women – ۱۷.۷ china – ۲.۳ japan – ۲.۹ variability is partly because of the different criteria and screening regimens magnitude of the problem india chennai hospital based universal screening – ۱۸.۹ had fpg ≥ ۱۲۶ and pppg ≥ ۱۴ . trivandrum – ۱۵ bangalore – ۱۲ erode – ۱۸.۸ chennai community based universal screning ۱۷.۸ in urban ۱۳.۸ in semi urban and ۹.۹ in rural areas. chennai .۵۶ mysore parthenon study ۶ maharashtra hospital based selective screening – ۷.۷ had gdm ۱۳.۹ had iggt. risk factors a family history of diabetes especially in first degree relatives prepregnancy weight ≥۱۱ of ideal body weight or body mass index over ۳ kg m۲ or significant weight gain in early adulthood between pregnancies or in early pregnancy age greater than ۲۵ years previous delivery of a baby greater than ۴.۱ kg personal history of abnormal glucose tolerance member of an ethnic group with higher than the background rate of type ۲ diabetes in most populations the background rate is approximately ۲ percent previous unexplained perinatal loss or birth of a malformed child maternal birthweight greater than ۴.۱ kg or less than ۶ pounds ۲.۷ kg glycosuria at the first prenatal visit polycystic ovary syndrome current use of glucocorticoids essential hypertension or pregnancy related hypertension maternal complications worsening retinopathy – ۱ new dr ۲ mild npdr and ۵۵ mod severe npdr progresses worsening proteinuria. gfr decline depends on preconception creatinine and proteinuria hypertension and cardiovascular disease neuropathy – no worsening gastroparesis nausea orthostatic dizziness can be worsened infection maternofetal complications macrosomia ۶۳ percent cesarean delivery ۵۶ percent preterm delivery ۴۲ percent preeclampsia ۱۸ percent respiratory distress syndrome ۱۷ percent congenital malformations ۵ percent perinatal mortality ۳ percent spontaneous abortion third trimester fetal deaths polyhydramnios preterm birth adverse neurodevelopmental outcome risk for type ۲ dm neonatal complications morbidity associated with preterm birth macrosomia ± birth injury shouldeer dystocia brachial plexus injury polycythemia and hyperviscosity hyperbilirubinemia cardiomyopathy hypoglycemia and other metabolic abnormalities hypocalcemia hypomagnesemia respiratory problems congenital anomalies congenital anomalies ۲ ۳rd cvs or cns – ۱۳ ۲ times common cardiac including great vessel anomalies most common central nervous system spina bifida anencephaly ۷.۲ skeletal cleft lip palate caudal regression syndrome genitourinary tract ureteric duplication gastrointestinal anorectal atresia skeletal and central nervous system caudal regression syndrome neural tube defects excluding anencephaly anencephaly with or without herniation of neural elements microcephaly cardiac transposition of the great vessels with or without ventricular ventricular septal defects coarctation of the aorta with or without ventricular septal defects or patent ductus arteriosus atrial septal defects cardiomegaly renal anomalies hydronephrosis renal agenesis ureteral duplication gastrointestinal duodenal atresia anorectal atresia small left colon syndrome caudal regression syndrome caudal regression syndrome whom to screen no consensus recommended screening ranges from selective screening of average and high risk individuals to universal diagnostic testing of the entire population dependent on the risk of diabetes in the population. risk stratification based on certain variables low risk no screening average risk at ۲۴ ۲۸ weeks high risk as soon as possible to satisfy …

کلمات کلیدی پرکاربرد در این اسلاید پاورپوینت: –, pregnancy, ., insulin, glucose, risk, percent, syndrome, gdm, screening, metabolism, type,

این فایل پاورپوینت شامل 65 اسلاید و به زبان انگلیسی و حجم آن 1.35 مگا بایت است. نوع قالب فایل ppt بوده که با این لینک قابل دانلود است. این مطلب برگرفته از سایت زیر است و مسئولیت انتشار آن با منبع اصلی می باشد که در تاریخ 2019/05/16 05:16:58 استخراج شده است.

https://www.cmcendovellore.org/downloads/diabetes-pregnancy.ppt

  • جهت آموزش های پاورپوینت بر روی اینجا کلیک کنید.
  • جهت دانلود رایگان قالب های حرفه ای پاورپوینت بر روی اینجا کلیک کنید.

رفتن به مشاهده اسلاید در بالای صفحه


دیدگاهتان را بنویسید

نشانی ایمیل شما منتشر نخواهد شد. بخش‌های موردنیاز علامت‌گذاری شده‌اند *