bone ، osteoid و tumour…

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نوع زبان: انگلیسی حجم: 0.07 مگا بایت
نوع فایل: اسلاید پاورپوینت تعداد اسلایدها: 25 صفحه
سطح مطلب: نامشخص پسوند فایل: ppt
گروه موضوعی: زمان استخراج مطلب: 2019/05/17 12:55:47

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عبارات مهم استفاده شده در این مطلب

عبارات مهم استفاده شده در این مطلب

bone, fracture, osteoid, ‘s, tumour, fibrous, osteoma, disease, tissue, end, –, osteoporosis,

توجه: این مطلب در تاریخ 2019/05/17 12:55:47 به صورت خودکار از فضای وب آشکار توسط موتور جستجوی پاورپوینت جمع آوری شده است و در صورت اعلام عدم رضایت تهیه کننده ی آن، طبق قوانین سایت از روی وب گاه حذف خواهد شد. این مطلب از وب سایت زیر استخراج شده است و مسئولیت انتشار آن با منبع اصلی است.

https://www.fnhk.cz/fs384/musculoskeletalpathology01web.ppt

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عبارات پرتکرار و مهم در این اسلاید عبارتند از: bone, fracture, osteoid, ‘s, tumour, fibrous, osteoma, disease, tissue, end, –, osteoporosis,

مشاهده محتوای متنیِ این اسلاید ppt

مشاهده محتوای متنیِ این اسلاید ppt

musculoskeletal pathology part i bones bone diseases metabolic bone diseases osteoporosis osteomalacia and rickets fibrous osteodystrophy paget’s disease fractures inflammations osteomyelitis tuberculous osteomyelitis tumours primary secondary metastatic osteoporosis absolute decline in the bone mass bone atrophy pathogenesis excessive amount of osteoclastic resorption or impairment of osteoblast mediated bone formation or both primary osteoporosis type i osteoporosis postmenopausal ↓oestrogen → ↑bone resorption ↑number and activity of osteoclasts type ii osteoporosis senile individuals older than ۷ years impairment of bone formation failure to replace resorbed bone secondary osteoporosis cushing’s syndrome hyperthyreoidism hypogonadism early oophorectomy malnutrition subtotal gastrectomy immobilisation hemiplegia paraplegia treatment with glucocorticoids or certain anticonvulsants clinical features fractures as a result of minimal trauma hip fractures compressive fractures of the vertebral bodies – kyphosis dowager’s hump fractures of the distal radius colles’ fracture morphology thinning of individual trabecula loss of normal trabecular network mineralisation is normal osteomalacia and rickets rachitis defective mineralisation of osteoid matrix lack of vitamin d low dietary intake lack of sunlight small gut malapsorption chronic liver and or renal diseases impaired hydroxylation of vitamin d precursors hypophosphataemia much less common resistance to treatment with vitamin d hereditary abnormality of phosphate transport by the renal tubular epithelium tumour associated effect of certain mesenchymal neoplasms osteomalacia adults with closed epiphyses normal bone mass but increase in amount of osteoid more than ۲ of the trabecular surface deformities kyphoscoliosis contracted pelvis limb curvature fractures rickets children epiphyses still open and active failure of matrix mineralisation disturbance of endochondral ossification very poor formation of zone of provisional calcification marked enlargement of zone of hypertrophic cartilage deformities craniotabes rhachitica caput quadratum frontal and parietal bossing bulging of the costochondral junctions „rickety rosary fibrous osteodystrophy severe hyperparathyreoidism especially primary excessive osteoclastic resorption of bone outline of bony trabeculae shows deep indentations filled with numerous osteoclasts and connective tissue in severe cases occur so called brown tumors a misnomer areas in which trabecular bone has been completely eroded away and replaced by vascular fibrous tissue with many osteoclasts and hemosiderin depositions brown color – should not to be confused with true giant cell tumor uf bone clinical features bone pain pathological fractures occasionally hypercalcaemia paget’s disease of bone osteitis deformans chronic condition enlargement and deformities of affected bones individuals over the age of ۴۵ years frequency increases with advancing age only one bone or many bones polyostotic form sites skull pelvis spine aetiology unknown some osteoclasts contain intranuclear inclusions resembling the nucleocapsid structure of paramyxoviruses → viral infection suggested but attempts to isolate virus failed macroscopic features bones enlarged and thickened thick calvarium and softer deformities – bowing of lower limbs narrowing of the foramina of cranial nerves neuropathies deafness microscopic features repeated episodes of intense bone resorption and subsequent reparation osteolytic phase mixed or osteoblastic phase osteosclerotic or burned out phase mosaic like appearance due to numerous cement lines greater risk of developing osteosarcoma fractures breaks in the continuity of bone infraction incomplete fracture pathological fracture bone altered by some previous lesion osteoporosis cysts tumours only minor trauma or spontaneously fracture healing haematoma between severed ends of bone necrosis of bone marrow organization of haematoma with granulation tissue connection of bone ends by fibrous tissue fibrous callus woven bone and cartilage in fibrous tissue provisional callus gradual remodelling to mature lamellar bone definite callus impaired fracture healing infection poor immobilization metabolic bone diseases exuberant callus with excessive cartilage formation sometimes non union of bone ends pseudo arthrosis osteomyelitis infection mediated inflammation of bone pus forming organisms staphylococci streptococci … infection may penetrate bone directly compound fractures operations via the bloodstream from distant primary focus haematogeneous osteomyelitis morphology acute pyogenic inflammation of bone marrow – rise in interstitial pressure compromises the local blood supply – ischaemic necrosis of cancellous bone spread of inflammation to the subperiosteal space subperiosteal abscess – periosteal blood vessels shear off ischaemic necrosis of cortical bone perforation of the periosteum – spread through soft tissues and skin forming a sinus track portions of dead bone can be discharged onto the skin complications sepsis amyloidosis pyogenic arthritis squamous carcinoma of the skin after many years tuberculous osteomyelitis children blood borne infection long bones vertebrae bone destruction associated with granulomatous lesion with caseation tuberculosis of the spine pott’s disease sharp anterior angulation of the spine kyphosis caseation may spread to the paravertebral soft tissues and track down along the psoas muscle to form an inguinal subcutaneous mass bone metastases much more common than the primary bone tumours usually multiple osteolytic bone destruction osteoblastic reactive bone formation tumors commonly producing bone metastases prostatic carcinoma breast carcinoma renal cell carcinoma carcinoma of stomach thyroid carcinoma primary bone tumours less common usually solitary features of considerable diagnostic value age of the patient site of the lesion classification simplified bone forming tumours osteoma osteoid osteoma osteoblastoma osteosarcoma cartilage forming tumours osteochondroma chondroma chondrosarcoma other tumours giant cell tumor of bone ewing’s sarcoma tumour like lesions fibrous dysplasia bone cysts osteoma benign lesion dense mature lamellar bone skull facial bones paranasal sinuses orbit macro usually small bony outgrowth micro mature lamellar bone may be a component of gardner’s colonic polyposis syndrome osteoid osteoma benign osteoblastic lesion young people ۵ ۲۴ years male predominance clinical features quite severe pain long bones femur tibia ends of the shafts morphology sharply outlined osteolytic defect nidus less than ۱ cm in diameter surrounded by rim of sclerotic bone nidus composed of haphazardly oriented trabeculae of woven bone and highly vacularized fibrous stroma trabeculae lined by plump osteoblasts and rare osteoklasts osteoblastoma rare closely related to osteoid osteoma but larger than ۱cm giant osteoid osteoma and generally lacking the reactive rim of dense sclerotic bone age similar to that of osteoid osteoma spine small bones of the hands and feet microscopical appearance very similar to osteoid osteoma trabeculae of woven bone lined by plump osteoblasts and osteoclasts vascularized fibrous stroma benign but examples showing atypical osteoblast and a poorly demarcated edge tend to recur although not metastasise aggressive osteoblastoma osteosarcoma most common primary malignant neoplasm of bone peak incidence between ۱ ۲۵ years in middle aged and elderly people associated with paget’s disease predominance of males metaphyseal region of the long bones lower end of femur upper end of tibia upper end of humerus arises from within medullary cavity and invades the cortical bone elevates the periosteum codman’s triangle and penetrates into soft tissues microscopic features bone or osteoid formation …

کلمات کلیدی پرکاربرد در این اسلاید پاورپوینت: bone, fracture, osteoid, ‘s, tumour, fibrous, osteoma, disease, tissue, end, –, osteoporosis,

این فایل پاورپوینت شامل 25 اسلاید و به زبان انگلیسی و حجم آن 0.07 مگا بایت است. نوع قالب فایل ppt بوده که با این لینک قابل دانلود است. این مطلب برگرفته از سایت زیر است و مسئولیت انتشار آن با منبع اصلی می باشد که در تاریخ 2019/05/17 12:55:47 استخراج شده است.

https://www.fnhk.cz/fs384/musculoskeletalpathology01web.ppt

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